Introduction
This paper discusses the physiological reason for Simon's breathlessness by reference to his ability to have homeostasis preserved while fighting infection and maintaining the respiratory system in proper function.
Question 1
Simon's breathlessness can be ascribed to the loss of homeostasis in his respiratory system. Homeostasis refers to the tendency of the human body to regulate itself to maintain its normal state and internal environment, which encompasses maintaining a stable level of oxygen and carbon dioxide for the cells to use (Meizlish et al., 2021). When Simon's oxygen saturation stands at 94% on air, that translates into a modest drop from optimal range (95-100%) indicating the body is not after all oxygenating tissues effectively. The function of the respiratory system is gas exchange where oxygen is absorbed and carbon dioxide is expelled. The productive cough alongside Simon's elevated temperature of 38.2°C registers that possibly there is an infection currently within his respiratory tract.
It leads to swelling and excessive production of mucus, which causes airways to narrow; consequently, it blocks airflow (Li & Tang, 2021). Consequently, Simon's lungs cannot breathe sufficient oxygen, while carbon dioxide cannot be expelled from the system; therefore, this condition can significantly affect homeostasis, creating a response. This condition was opposed by increasing Simon's respiratory rate by 24 breaths/min to meet the body's demands for oxygen while eliminating excess carbon dioxide. This is not entirely effective due to the presence of inflammation within the airways, and Simon soon finds himself breathless because of the body's attempt to regain its balance.
Conclusion
In general, symptoms that characterize Simon indicate the disruptions from homeostasis that occur as a result of respiratory infections culminating in compensatory mechanisms which appear as shortness of breath. It is by understanding these mechanisms that one can achieve efficient diagnosis and treatment.